Misfolded α-synuclein and toll-like receptors: therapeutic targets for Parkinson's disease
- 31 January 2012
- journal article
- review article
- Published by Elsevier BV in Parkinsonism & Related Disorders
- Vol. 18 (0 1), S17-S20
- https://doi.org/10.1016/s1353-8020(11)70008-6
Abstract
No abstract availableThis publication has 20 references indexed in Scilit:
- Toll-Like Receptors: New Players in Myocardial Ischemia/Reperfusion InjuryAntioxidants and Redox Signaling, 2011
- Common genetic variation in the HLA region is associated with late-onset sporadic Parkinson's diseaseNature Genetics, 2010
- Assessing activation states in microglia.CNS & Neurological Disorders - Drug Targets, 2010
- Neuroinflammation in the living brain of Parkinson's diseaseParkinsonism & Related Disorders, 2009
- Targeted Overexpression of Human α-Synuclein Triggers Microglial Activation and an Adaptive Immune Response in a Mouse Model of Parkinson DiseaseJournal of Neuropathology and Experimental Neurology, 2008
- α-Synuclein Misfolding and Neurodegenerative DiseasesCurrent Protein & Peptide Science, 2008
- Nongenetic causes of Parkinson’s diseaseJournal Of Neural Transmission-Supplement, 2006
- Aggregated α‐synuclein activates microglia: a process leading to disease progression in Parkinson's diseaseThe FASEB Journal, 2005
- Tumor necrosis factor gene polymorphisms in patients with sporadic Parkinson's diseaseNeuroscience Letters, 2001
- The Single Intranigral Injection of LPS as a New Model for Studying the Selective Effects of Inflammatory Reactions on Dopaminergic SystemNeurobiology of Disease, 2000