Adverse ventricular remodeling and exacerbated NOS uncoupling from pressure-overload in mice lacking the β3-adrenoreceptor
- 30 November 2009
- journal article
- research article
- Published by Elsevier BV in Journal of Molecular and Cellular Cardiology
- Vol. 47 (5), 576-585
- https://doi.org/10.1016/j.yjmcc.2009.06.005
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- Cardiac Myocyte–Specific Expression of Inducible Nitric Oxide Synthase Protects Against Ischemia/Reperfusion Injury by Preventing Mitochondrial Permeability TransitionCirculation, 2008
- Reversal of Cardiac Hypertrophy and Fibrosis From Pressure Overload by TetrahydrobiopterinCirculation, 2008
- High-Dose Folic Acid Pretreatment Blunts Cardiac Dysfunction During Ischemia Coupled to Maintenance of High-Energy Phosphates and Reduces Postreperfusion InjuryCirculation, 2008
- Role of Oxidative Stress in Cardiac Hypertrophy and RemodelingHypertension, 2007
- Tetrahydrobiopterin and Cardiovascular DiseaseArteriosclerosis, Thrombosis, and Vascular Biology, 2006
- Induction of β3-Adrenergic Receptor Functional Expression following Chronic Stimulation with Noradrenaline in Neonatal Rat CardiomyocytesJournal of Pharmacology and Experimental Therapeutics, 2005
- Endogenous β3-adrenoreceptor activation contributes to left ventricular and cardiomyocyte dysfunction in heart failureAmerican Journal of Physiology-Heart and Circulatory Physiology, 2004
- Disruption of Leptin Signaling Contributes to Cardiac Hypertrophy Independently of Body Weight in MiceCirculation, 2003
- Potential Use of β3‐Adrenoceptor Antagonists in Heart Failure TherapyCardiovascular Drug Reviews, 2002
- Comparison of the Lipolytic Effects of Norepinephrine and BRL 37344 in Rat Brown and White AdipocytesObesity Research, 1994