Prevention of NKT Cell Activation Accelerates Cutaneous Wound Closure and Alters Local Inflammatory Signals
- 30 November 2011
- journal article
- Published by Elsevier BV in Journal of Surgical Research
- Vol. 171 (1), 361-373
- https://doi.org/10.1016/j.jss.2010.03.030
Abstract
No abstract availableKeywords
This publication has 60 references indexed in Scilit:
- FcgRIII engagement provides activating signals to NKT cells in antibody-induced joint inflammationJCI Insight, 2006
- Glucocorticoids suppress bone formation via the osteoclastJCI Insight, 2006
- Activated protein C prevents inflammation yet stimulates angiogenesis to promote cutaneous wound healingWound Repair and Regeneration, 2005
- Trafficking machinery of NKT cells: shared and differential chemokine receptor expression among Vα24+Vβ11+ NKT cell subsets with distinct cytokine-producing capacityBlood, 2002
- CD1d-dependent macrophage-mediated clearance of Pseudomonas aeruginosa from lungNature Medicine, 2002
- CD1d on antigen-transporting APC and splenic marginal zone B cells promotes NKT cell-dependent toleranceEuropean Journal of Immunology, 2002
- A cost analysis of diabetic lower-extremity ulcers.Diabetes Care, 2000
- Cutaneous Wound HealingThe New England Journal of Medicine, 1999
- THE CD1 SYSTEM: Antigen-Presenting Molecules for T Cell Recognition of Lipids and GlycolipidsAnnual Review of Immunology, 1999
- The CD69 receptor: a multipurpose cell-surface trigger for hematopoietic cellsImmunology Today, 1994