Class IC antiarrhythmic drugs, flecainide and pilsicainide, produce ST segment elevation simulating inferior myocardial ischemia.
- 1 August 1998
- journal article
- case report
- Published by Wiley in Journal of Cardiovascular Electrophysiology
- Vol. 9 (8), 855-858
- https://doi.org/10.1111/j.1540-8167.1998.tb00125.x
Abstract
No abstract availableKeywords
This publication has 11 references indexed in Scilit:
- β-Adrenergic Augmentation of Flecainide-Induced Conduction Slowing in Canine Purkinje FibersCirculation, 1997
- Discrepant drug action of disopyramide on ECG abnormalities and induction of ventricular arrhythmias in a patient with Brugada syndromeJournal of Electrocardiology, 1997
- Safety and Efficacy of Oral Flecainide Acetate in Patients with Cardiac Arrhythmias.Japanese Heart Journal, 1997
- Conversion of Recent-Onset Atrial Fibrillation by a Single Oral Dose of Pilsicainide (Pilsicainide Suppression Trial on Atrial Fibrillation)The American Journal of Cardiology, 1996
- Autonomic and antiarrhythmic drug modulation of ST segment elevation in patients with Brugada syndromeJournal of the American College of Cardiology, 1996
- Mechanisms of Negative Inotropic Effects of Class Ic Antiarrhythmic Agents: Comparative Study of the Effects of Flecainide and Pilsicainide on Intracellular Calcium Handling in Dog Ventricular MyocardiumJournal of Cardiovascular Pharmacology, 1996
- Electrophysiological Effects of MS‐551 in Humans: A Class III Antiarrhythmic AgentPacing and Clinical Electrophysiology, 1995
- Ischemia enhances use-Dependent sodium channel blockade by pilsicainide, a class IC antiarrhythmic agentJournal of the American College of Cardiology, 1994
- Flecainide-induced arrhythmia in canine ventricular epicardium. Phase 2 reentry?Circulation, 1993
- Heterogeneity within the ventricular wall. Electrophysiology and pharmacology of epicardial, endocardial, and M cells.Circulation Research, 1991