Effects of the NIK aly Mutation on NF-κB Activation by the Epstein-Barr Virus Latent Infection Membrane Protein, Lymphotoxin β Receptor, and CD40
Open Access
- 1 January 2001
- journal article
- Published by Elsevier BV in Journal of Biological Chemistry
- Vol. 276 (18), 14602-14606
- https://doi.org/10.1074/jbc.c100103200
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- The NF-κB-inducing Kinase Induces PC12 Cell Differentiation and Prevents ApoptosisJournal of Biological Chemistry, 2000
- Negative Regulation of the Nuclear Factor κB-inducing Kinase by a cis-Acting DomainPublished by Elsevier BV ,2000
- The Distinct Roles of TRAF2 and RIP in IKK Activation by TNF-R1Immunity, 2000
- Lineage-Restricted Function of Nuclear Factor κB–Inducing Kinase (Nik) in Transducing Signals via Cd40The Journal of Experimental Medicine, 2000
- Epstein-Barr virus transformation: involvement of latent membrane protein 1-mediated activation of NF-κBOncogene, 1999
- Positive and Negative Regulation of IκB Kinase Activity Through IKKβ Subunit PhosphorylationScience, 1999
- Activation of NF-κB by RANK Requires Tumor Necrosis Factor Receptor-associated Factor (TRAF) 6 and NF-κB-inducing KinasePublished by Elsevier BV ,1999
- The Lymphotoxin β Receptor Controls Organogenesis and Affinity Maturation in Peripheral Lymphoid TissuesImmunity, 1998
- Nuclear Factor (NF)-κB2 (p100/p52) Is Required for Normal Splenic Microarchitecture and B Cell–mediated Immune ResponsesThe Journal of Experimental Medicine, 1998
- The MAP kinase Fus3 associates with and phosphorylates the upstream signaling component Ste5.Genes & Development, 1994