The oncogenic serine/threonine kinase Pim-1 directly phosphorylates and activates the G2/M specific phosphatase Cdc25C
- 31 March 2006
- journal article
- Published by Elsevier BV in The International Journal of Biochemistry & Cell Biology
- Vol. 38 (3), 430-443
- https://doi.org/10.1016/j.biocel.2005.10.010
Abstract
No abstract availableKeywords
This publication has 43 references indexed in Scilit:
- The Oncogenic Serine/Threonine Kinase Pim-1 Phosphorylates and Inhibits the Activity of Cdc25C-associated Kinase 1 (C-TAK1)Journal of Biological Chemistry, 2004
- Pim‐1 kinase promotes inactivation of the pro‐apoptotic Bad protein by phosphorylating it on the Ser112 gatekeeper siteFEBS Letters, 2004
- Pim-1 associates with protein complexes necessary for mitosisChromosoma, 2002
- Pim serine/threonine kinases regulate the stability of Socs-1 proteinProceedings of the National Academy of Sciences of the United States of America, 2002
- Oral cancer in vivo gene expression profiling assisted by laser capture microdissection and microarray analysisOncogene, 2001
- Prolactin Regulation of pim-1 Expression: Positive and Negative Promoter ElementsEndocrinology, 1999
- Ectopic Expression of Cdc25A Accelerates the G1/S Transition and Leads to Premature Activation of Cyclin E- and Cyclin A-Dependent KinasesMolecular and Cellular Biology, 1999
- Pim-2 transgene induces lymphoid tumors, exhibiting potent synergy with c-mycOncogene, 1997
- Rapid induction of pim-1 expression by prolactin and interleukin-2 in rat Nb2 lymphoma cellsEndocrinology, 1995
- Murine leukemia virus-induced T-cell lymphomagenesis: Integration of proviruses in a distinct chromosomal regionCell, 1984