Neuroprotective effect of Nrf2/ARE activators, CDDO ethylamide and CDDO trifluoroethylamide, in a mouse model of amyotrophic lateral sclerosis
- 1 July 2011
- journal article
- Published by Elsevier BV in Free Radical Biology & Medicine
- Vol. 51 (1), 88-96
- https://doi.org/10.1016/j.freeradbiomed.2011.03.027
Abstract
No abstract availableKeywords
This publication has 59 references indexed in Scilit:
- Triterpenoids CDDO-ethyl amide and CDDO-trifluoroethyl amide improve the behavioral phenotype and brain pathology in a transgenic mouse model of Huntington's diseaseFree Radical Biology & Medicine, 2010
- Stress-Activated Cap'n'collar Transcription Factors in Aging and Human DiseaseScience Signaling, 2010
- Astrocyte-Specific Overexpression of Nrf2 Protects Striatal Neurons from Mitochondrial Complex II InhibitionToxicological Sciences, 2010
- Mutant SOD1 in neuronal mitochondria causes toxicity and mitochondrial dynamics abnormalitiesHuman Molecular Genetics, 2009
- Neuroprotective Effects of the Triterpenoid, CDDO Methyl Amide, a Potent Inducer of Nrf2-Mediated TranscriptionPLOS ONE, 2009
- Triterpenoid CDDO‐methylamide improves memory and decreases amyloid plaques in a transgenic mouse model of Alzheimer’s diseaseJournal of Neurochemistry, 2009
- Nrf2 Activation in Astrocytes Protects against Neurodegeneration in Mouse Models of Familial Amyotrophic Lateral SclerosisJournal of Neuroscience, 2008
- Keap1/Nrf2 Signaling Regulates Oxidative Stress Tolerance and Lifespan in DrosophilaDevelopmental Cell, 2008
- DJ-1, a cancer- and Parkinson's disease-associated protein, stabilizes the antioxidant transcriptional master regulator Nrf2Proceedings of the National Academy of Sciences of the United States of America, 2006
- Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT MethodMethods, 2001