Modeling of the Role of a Bax-Activation Switch in the Mitochondrial Apoptosis Decision
- 15 June 2007
- journal article
- Published by Elsevier BV in Biophysical Journal
- Vol. 92 (12), 4304-4315
- https://doi.org/10.1529/biophysj.106.099606
Abstract
No abstract availableKeywords
Funding Information
- National Natural Science Foundation of China (20477016, 30571538)
- Natural Science Foundation of Jiangsu Province
This publication has 51 references indexed in Scilit:
- Stochastic models for regulatory networks of the genetic toggle switchProceedings of the National Academy of Sciences, 2006
- Auto-activation of the Apoptosis Protein Bax Increases Mitochondrial Membrane Permeability and Is Inhibited by Bcl-2Published by Elsevier BV ,2006
- Rapamycin induces apoptosis of JN-DSRCT-1 cells by increasing the Bax : Bcl-xL ratio through concurrent mechanisms dependent and independent of its mTOR inhibitory activityOncogene, 2005
- Bax interacts with the voltage-dependent anion channel and mediates ethanol-induced apoptosis in rat hepatocytesAmerican Journal of Physiology-Gastrointestinal and Liver Physiology, 2004
- Bifurcation analysis of the regulatory modules of the mammalian G1/S transitionBioinformatics, 2004
- Black tea protects immunocytes from tumor-induced apoptosis by changing Bcl-2/Bax ratioCancer Letters, 2004
- Generation of a novel apoptosis-resistant hepatoma cell lineJournal of Bioscience and Bioengineering, 2003
- Spatial and temporal changes in Bax subcellular localization during anoikisThe Journal of cell biology, 2003
- BCL-2 Selectively Interacts with the BID-induced Open Conformer of BAK, Inhibiting BAK Auto-oligomerizationJournal of Biological Chemistry, 2003
- Inhibition of Bid-induced Apoptosis by Bcl-2Published by Elsevier BV ,2003