DNA-PKcs and ATM co-regulate DNA double-strand break repair
- 6 August 2009
- journal article
- Published by Elsevier BV in DNA Repair
- Vol. 8 (8), 920-929
- https://doi.org/10.1016/j.dnarep.2009.05.006
Abstract
No abstract availableKeywords
This publication has 72 references indexed in Scilit:
- Phosphorylation in the serine/threonine 2609–2647 cluster promotes but is not essential for DNA-dependent protein kinase-mediated nonhomologous end joining in human whole-cell extractsNucleic Acids Research, 2007
- trans Autophosphorylation at DNA-Dependent Protein Kinase's Two Major Autophosphorylation Site Clusters Facilitates End Processing but Not End JoiningMolecular and Cellular Biology, 2007
- Autophosphorylation of DNA-PKCS regulates its dynamics at DNA double-strand breaksThe Journal of cell biology, 2007
- Homologous recombination repair is regulated by domains at the N- and C-terminus of NBS1 and is dissociated with ATM functionsOncogene, 2007
- Ataxia Telangiectasia Mutated (ATM) Is Essential for DNA-PKcs Phosphorylations at the Thr-2609 Cluster upon DNA Double Strand BreakOnline Journal of Public Health Informatics, 2007
- The rad51-K191R ATPase-Defective Mutant Is Impaired forPresynaptic Filament FormationMolecular and Cellular Biology, 2006
- ATM stabilizes DNA double-strand-break complexes during V(D)J recombinationNature, 2006
- Small molecule–based reversible reprogramming of cellular lifespanNature Chemical Biology, 2006
- γH2AX and MDC1: Anchoring the DNA-damage-response machinery to broken chromosomesDNA Repair, 2006
- Genome architecture, rearrangements and genomic disordersTrends in Genetics, 2002