Endosomal Acidification and Activation of NADPH Oxidase Isoforms Are Upstream Events in Hyperosmolarity-induced Hepatocyte Apoptosis
Open Access
- 1 August 2006
- journal article
- Published by Elsevier BV
- Vol. 281 (32), 23150-23166
- https://doi.org/10.1074/jbc.m601451200
Abstract
No abstract availableThis publication has 74 references indexed in Scilit:
- Role of sphingomyelinase and ceramide in modulating rafts: do biophysical properties determine biologic outcome?FEBS Letters, 2002
- Hyperosmolarity triggers CD95 membrane trafficking and sensitizes rat hepatocytes toward CD95L-induced apoptosisHepatology, 2002
- De Novo Sphingolipid Biosynthesis: A Necessary, but Dangerous, PathwayJournal of Biological Chemistry, 2002
- Correction of CFTR malfunction and stimulation of Ca2+ -activated Cl− channels restore HCO 3− secretion in cystic fibrosis bile ductular cellsHepatology, 2002
- Enzymes of Sphingolipid Metabolism: From Modular to Integrative SignalingBiochemistry, 2001
- Natural Ceramide Reverses Fas Resistance of Acid Sphingomyelinase −− HepatocytesPublished by Elsevier BV ,2001
- Diverse functions of JNK signaling and c-Jun in stress response and apoptosisOncogene, 1999
- Osmotic Induction of Signaling Cascades: Role in Regulation of Cell FunctionBiochemical and Biophysical Research Communications, 1999
- Alkalinization of acidic cellular compartments following cell swellingFEBS Letters, 1994
- Inhibitory effect of neopterin on NADPH‐dependent superoxide‐generating oxidase of rat peritoneal macrophagesFEBS Letters, 1993