Decreased cellular cholesterol efflux is a common cause of familial hypoalphalipoproteinemia: role of the ABCA1 gene mutations
- 1 October 2000
- journal article
- Published by Elsevier BV in Atherosclerosis
- Vol. 152 (2), 457-468
- https://doi.org/10.1016/s0021-9150(99)00498-0
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Protein kinase C-α levels are inversely associated with growth rate in cultured human dermal fibroblastsJournal of Dermatological Science, 1998
- Decreased Cholesterol Efflux from Fibroblasts of a Patient without Tangier Disease, but with Markedly Reduced High Density Lipoprotein Cholesterol LevelsJournal of Clinical Endocrinology & Metabolism, 1998
- Decreased Reverse Cholesterol Transport from Tangier Disease FibroblastsArteriosclerosis, Thrombosis, and Vascular Biology, 1997
- Severe Familial HDL Deficiency in French-Canadian KindredsArteriosclerosis, Thrombosis, and Vascular Biology, 1995
- HDL-Mediated Efflux of Intracellular Cholesterol Is Impaired in Fibroblasts From Tangier Disease PatientsArteriosclerosis, Thrombosis, and Vascular Biology, 1995
- Familial hypoalphalipoproteinemia in premature coronary artery disease.Arteriosclerosis and Thrombosis: A Journal of Vascular Biology, 1993
- Familial HDL deficiency due to marked hypercatabolism of normal apoA-I.Arteriosclerosis and Thrombosis: A Journal of Vascular Biology, 1993
- In vivo metabolism of a mutant form of apolipoprotein A-I, apo A-IMilano, associated with familial hypoalphalipoproteinemia.JCI Insight, 1993
- Prevalence of risk factors in men with premature coronary artery diseaseThe American Journal of Cardiology, 1991
- Regulation of the mevalonate pathwayNature, 1990