Mitochondria, oxidative metabolism and cell death in stroke
Top Cited Papers
- 31 January 2010
- journal article
- review article
- Published by Elsevier BV in Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
- Vol. 1802 (1), 80-91
- https://doi.org/10.1016/j.bbadis.2009.09.003
Abstract
No abstract availableKeywords
This publication has 110 references indexed in Scilit:
- Mechanisms of ischemic brain damageNeuropharmacology, 2008
- Bid-induced release of AIF from mitochondria causes immediate neuronal cell deathCell Death & Differentiation, 2008
- Proteolysis of AKAP121 regulates mitochondrial activity during cellular hypoxia and brain ischaemiaThe EMBO Journal, 2008
- Mechanisms of impaired mitochondrial energy metabolism in acute and chronic neurodegenerative disordersJournal of Neuroscience Research, 2007
- Influence of duration of focal cerebral ischemia and neuronal nitric oxide synthase on translocation of apoptosis-inducing factor to the nucleusNeuroscience, 2007
- Apoptosis-inducing factor mediates poly(ADP-ribose) (PAR) polymer-induced cell deathProceedings of the National Academy of Sciences of the United States of America, 2006
- Cyclophilin D is a component of mitochondrial permeability transition and mediates neuronal cell death after focal cerebral ischemiaProceedings of the National Academy of Sciences of the United States of America, 2005
- Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell deathNature, 2005
- Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell deathNature, 2005
- The ADP/ATP translocator is not essential for the mitochondrial permeability transition poreNature, 2004