Calcium-calmodulin dependent protein kinase II (CaMKII): A main signal responsible for early reperfusion arrhythmias
- 31 December 2011
- journal article
- research article
- Published by Elsevier BV in Journal of Molecular and Cellular Cardiology
- Vol. 51 (6), 936-944
- https://doi.org/10.1016/j.yjmcc.2011.08.010
Abstract
No abstract availableKeywords
This publication has 48 references indexed in Scilit:
- Ryanodine Receptor Phosphorylation by Calcium/Calmodulin-Dependent Protein Kinase II Promotes Life-Threatening Ventricular Arrhythmias in Mice With Heart FailureCirculation, 2010
- Transient Ca2+ depletion of the sarcoplasmic reticulum at the onset of reperfusionCardiovascular Research, 2009
- Ca/Calmodulin Kinase II Differentially Modulates Potassium CurrentsCirculation: Arrhythmia and Electrophysiology, 2009
- Increased intracellular Ca2+and SR Ca2+load contribute to arrhythmias after acidosis in rat heart. Role of Ca2+/calmodulin-dependent protein kinase IIAmerican Journal of Physiology-Heart and Circulatory Physiology, 2008
- A Dynamic Pathway for Calcium-Independent Activation of CaMKII by Methionine OxidationCell, 2008
- Ca2+/calmodulin kinase II increases ryanodine binding and Ca2+-induced sarcoplasmic reticulum Ca2+ release kinetics during β-adrenergic stimulationJournal of Molecular and Cellular Cardiology, 2007
- CaMKII inhibition targeted to the sarcoplasmic reticulum inhibits frequency-dependent acceleration of relaxation and Ca2+ current facilitationJournal of Molecular and Cellular Cardiology, 2007
- Ca2+/calmodulin-dependent protein kinase II regulates cardiac Na+ channelsJCI Insight, 2006
- Stabilization of cardiac ryanodine receptor prevents intracellular calcium leak and arrhythmiasProceedings of the National Academy of Sciences of the United States of America, 2006
- Time Course and Mechanisms of Phosphorylation of Phospholamban Residues in Ischemia-reperfused Rat Hearts. Dissociation of Phospholamban Phosphorylation PathwaysJournal of Molecular and Cellular Cardiology, 2002