The selenoprotein GPX4 is essential for mouse development and protects from radiation and oxidative damage insults
Top Cited Papers
- 11 January 2003
- journal article
- Published by Elsevier BV in Free Radical Biology & Medicine
- Vol. 34 (4), 496-502
- https://doi.org/10.1016/s0891-5849(02)01360-6
Abstract
No abstract availableKeywords
This publication has 21 references indexed in Scilit:
- Overexpression of PHGPx inhibits hydroperoxide-induced oxidation, NFκB activation and apoptosis and affects oxLDL-mediated proliferation of rabbit aortic smooth muscle cellsAtherosclerosis, 2000
- Mitochondrial phospholipid hydroperoxide glutathione peroxidase inhibits the release of cytochrome c from mitochondria by suppressing the peroxidation of cardiolipin in hypoglycaemia-induced apoptosisBiochemical Journal, 2000
- Dual Function of the Selenoprotein PHGPx During Sperm MaturationScience, 1999
- Antioxidant defenses in TNF-treated MCF-7 cells: selective increase in MnSODFree Radical Biology & Medicine, 1999
- ATM: from gene to functionHuman Molecular Genetics, 1998
- Expression of genes coding for antioxidant enzymes and heat shock proteins is altered in primary cultures of rat hepatocytesJournal of Cellular Physiology, 1996
- Dilated cardiomyopathy and neonatal lethality in mutant mice lacking manganese superoxide dismutaseNature Genetics, 1995
- The role of selenium peroxidases in the protection against oxidative damage of membranesChemistry and Physics of Lipids, 1987
- Toxicity determined in vitro by morphological alterations and neutral red absorptionToxicology Letters, 1985
- CLONAL GROWTH OF MAMMALIAN CELLS IN VITROThe Journal of Experimental Medicine, 1956