Sequential Molecular Changes during Multistage Pathogenesis of Small Peripheral Adenocarcinomas of the Lung
- 1 April 2008
- journal article
- research article
- Published by Elsevier BV in Journal of Thoracic Oncology
- Vol. 3 (4), 340-347
- https://doi.org/10.1097/jto.0b013e318168d20a
Abstract
No abstract availableKeywords
This publication has 42 references indexed in Scilit:
- Mucinous Differentiation Correlates with Absence of EGFR Mutation and Presence of KRAS Mutation in Lung Adenocarcinomas with Bronchioloalveolar FeaturesThe Journal of Molecular Diagnostics, 2007
- Polymorphisms, Mutations, and Amplification of the EGFR Gene in Non-Small Cell Lung CancersPLoS Medicine, 2007
- Lung adenocarcinomas induced in mice by mutant EGF receptors foundin human lung cancers respondto a tyrosine kinase inhibitor orto down-regulation of the receptorsGenes & Development, 2006
- Epidermal growth factor receptor gene mutation defines distinct subsets among small adenocarcinomas of the lungLung Cancer, 2006
- Somatic mutations of epidermal growth factor receptor signaling pathway in lung cancersInternational Journal of Cancer, 2006
- Erlotinib in Lung Cancer — Molecular and Clinical Predictors of OutcomeNew England Journal of Medicine, 2005
- KRAS Mutations and Primary Resistance of Lung Adenocarcinomas to Gefitinib or ErlotinibPLoS Medicine, 2005
- EGF receptor gene mutations are common in lung cancers from “never smokers” and are associated with sensitivity of tumors to gefitinib and erlotinibProceedings of the National Academy of Sciences of the United States of America, 2004
- EGFR Mutations in Lung Cancer: Correlation with Clinical Response to Gefitinib TherapyScience, 2004
- Activating Mutations in the Epidermal Growth Factor Receptor Underlying Responsiveness of Non–Small-Cell Lung Cancer to GefitinibNew England Journal of Medicine, 2004