Sex-dependent Effect of BAG1 in Ameliorating Motor Deficits of Huntington Disease Transgenic Mice
Open Access
- 1 June 2008
- journal article
- Published by Elsevier BV
- Vol. 283 (23), 16027-16036
- https://doi.org/10.1074/jbc.m710606200
Abstract
No abstract availableKeywords
This publication has 49 references indexed in Scilit:
- Impaired ubiquitin–proteasome system activity in the synapses of Huntington's disease miceThe Journal of cell biology, 2008
- Sex differences in behavior and striatal ascorbate release in the 140 CAG knock-in mouse model of Huntington's diseaseBehavioural Brain Research, 2007
- Sex differences in heat shock protein 72 expression and localization in rats following renal ischemia-reperfusion injuryAmerican Journal of Physiology-Renal Physiology, 2006
- Sexual dimorphism of the intracellular heat shock protein 72 responseJournal of Applied Physiology, 2006
- The Anti-Apoptotic, Glucocorticoid Receptor Cochaperone Protein BAG-1 Is a Long-Term Target for the Actions of Mood StabilizersJournal of Neuroscience, 2005
- Inclusion body formation reduces levels of mutant huntingtin and the risk of neuronal deathNature, 2004
- Huntingtin forms toxic NH2-terminal fragment complexes that are promoted by the age-dependent decrease in proteasome activityThe Journal of cell biology, 2003
- Bag1 is a regulator and marker of neuronal differentiationCell Death & Differentiation, 2002
- Aggregation of N-terminal huntingtin is dependent on the length of its glutamine repeatsHuman Molecular Genetics, 1998
- Aggregation of Huntingtin in Neuronal Intranuclear Inclusions and Dystrophic Neurites in BrainScience, 1997