Involvement of Bcl-2 and Bax in Photodynamic Therapy-mediated Apoptosis
Open Access
- 1 January 2001
- journal article
- Published by Elsevier BV
- Vol. 276 (18), 15481-15488
- https://doi.org/10.1074/jbc.m006920200
Abstract
No abstract availableThis publication has 39 references indexed in Scilit:
- Antisense Bcl-2 Retrovirus Vector Increases the Sensitivity of a Human Gastric Adenocarcinoma Cell Line to Photodynamic TherapyPhotochemistry and Photobiology, 1999
- Involvement of retinoblastoma (Rb) and E2F transcription factors during photodynamic therapy of human epidermoid carcinoma cells A431Oncogene, 1999
- Photodynamic Therapy-Induced Apoptosis in Lymphoma Cells: Translocation of Cytochrome c Causes Inhibition of Respiration as Well as Caspase ActivationBiochemical and Biophysical Research Communications, 1999
- Antisense Bcl-2 Retrovirus Vector Increases the Sensitivity of a Human Gastric Adenocarcinoma Cell Line to Photodynamic TherapyPhotochemistry and Photobiology, 1999
- Phthalocyanine 4 (Pc 4) Photodynamic Therapy of Human OVCAR-3 Tumor XenograftsPhotochemistry and Photobiology, 1999
- Association of Ceramide Accumulation with Photodynamic Treatment-Induced Cell DeathPhotochemistry and Photobiology, 1998
- Inhibition of Bax Channel-Forming Activity by Bcl-2Science, 1997
- The Induction of Partial Resistance to Photodynamic Therapy by the Protooncogene BCL‐2Photochemistry and Photobiology, 1996
- Molecular Ordering of the Cell Death Pathway: Bcl-2 AND Bcl-xL FUNCTION UPSTREAM OF THE CED-3-LIKE APOPTOTIC PROTEASESPublished by Elsevier BV ,1996
- Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programed cell deathCell, 1993