Pioglitazone reduces neointimal tissue proliferation after coronary stent implantation in patients with type 2 diabetes mellitus: an intravascular ultrasound scanning study
- 31 August 2003
- journal article
- clinical trial
- Published by Elsevier BV in American Heart Journal
- Vol. 146 (2), 366-E5
- https://doi.org/10.1016/s0002-8703(03)00146-7
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- The Prevention or Delay of Type 2 DiabetesDiabetes Care, 2002
- Myocardial infarction and prevalence of diabetes mellitus. Is increased casual blood glucose at admission a reliable criterion for the diagnosis of diabetes?Published by Oxford University Press (OUP) ,2001
- Troglitazone reduces neointimal tissue proliferation after coronary stent implantation in patients with non–insulin dependent diabetes mellitus: A serial intravascular ultrasound studyJournal of the American College of Cardiology, 2000
- Pioglitazone hydrochloride monotherapy improves glycemic control in the treatment of patients with type 2 diabetes: a 6-month randomized placebo-controlled dose-response study. The Pioglitazone 001 Study Group.Diabetes Care, 2000
- Hyperinsulinemia during oral glucose tolerance test is associated with increased neointimal tissue proliferation after coronary stent implantation in nondiabetic patients: A serial intravascular ultrasound studyJournal of the American College of Cardiology, 2000
- Effect of statin therapy on restenosis after coronary stent implantationThe American Journal of Cardiology, 2000
- PPARγ-Ligands Inhibit Migration Mediated by Multiple Chemoattractants in Vascular Smooth Muscle CellsJournal of Cardiovascular Pharmacology, 1999
- Pioglitazone Reduces Smooth Muscle Cell Density of Rat Carotid Arterial Intima Induced by Balloon CatheterizationHormone and Metabolic Research, 1997
- Troglitazone inhibits vascular smooth muscle cell growth and intimal hyperplasia.JCI Insight, 1996
- Variations in lipoproteins, hormones and blood glucose during the early acute phase of myocardial infarctionAtherosclerosis, 1988