Comparison of gene expression in HCT116 treatment derivatives generated by two different 5-fluorouracil exposure protocols
Open Access
- 1 January 2004
- journal article
- research article
- Published by Springer Science and Business Media LLC in Molecular Cancer
- Vol. 3 (1), 11
- https://doi.org/10.1186/1476-4598-3-11
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- Molecular characterizations of derivatives of HCT116 colorectal cancer cells that are resistant to the chemotherapeutic agent 5-fluorouracil.International Journal of Oncology, 2004
- Drug resistance reversal—are we getting closer?European Journal Of Cancer, 2003
- Introduction to resistance to anticancer agentsOncogene, 2003
- Role of hMLH1 promoter hypermethylation in drug resistance to 5‐fluorouracil in colorectal cancer cell linesInternational Journal of Cancer, 2003
- 5-Fluorouracil: mechanisms of action and clinical strategiesNature Reviews Cancer, 2003
- Induction of thymidylate synthase as a 5-fluorouracil resistance mechanismBiochimica et Biophysica Acta (BBA) - Molecular Basis of Disease, 2002
- Role of the hMLH1 DNA mismatch repair protein in fluoropyrimidine-mediated cell death and cell cycle responses.2001
- Mutations of transforming growth factor beta 1 type II receptor, BAX, and insulin-like growth factor II receptor genes in microsatellite unstable cell lines.2000
- Thymidylate synthase gene amplification in human colon cancer cell lines resistant to 5-fluorouracilBiochemical Pharmacology, 1995
- Effect of clinically modeled regimens on the growth response and development of resistance in human colon carcinoma cell linesBiochemical Pharmacology, 1995