Receptor for advanced glycation endproducts (RAGE) and vascular inflammation: insights into the pathogenesis of macrovascular complications in diabetes.
- 1 May 2002
- journal article
- review article
- Published by Springer Science and Business Media LLC in Current Atherosclerosis Reports
- Vol. 4 (3), 228-237
- https://doi.org/10.1007/s11883-002-0024-4
Abstract
No abstract availableThis publication has 49 references indexed in Scilit:
- Biochemistry and molecular cell biology of diabetic complicationsNature, 2001
- PPARγ and AtherosclerosisArteriosclerosis, Thrombosis, and Vascular Biology, 2001
- High-glucose-induced nuclear factor κB activation in vascular smooth muscle cellsCardiovascular Research, 2000
- Receptor for Advanced Glycation End Products (RAGE)-mediated Neurite Outgrowth and Activation of NF-κB Require the Cytoplasmic Domain of the Receptor but Different Downstream Signaling PathwaysOnline Journal of Public Health Informatics, 1999
- RAGE Mediates a Novel Proinflammatory Axis: A Central Cell Surface Receptor for S100/Calgranulin PolypeptidesCell, 1999
- Intensive blood-glucose control with sulphonylureas or insulin compared with conventional treatment and risk of complications in patients with type 2 diabetes (UKPDS 33)The Lancet, 1998
- Amelioration of Vascular Dysfunctions in Diabetic Rats by an Oral PKC β InhibitorScience, 1996
- Spontaneous Hypercholesterolemia and Arterial Lesions in Mice Lacking Apolipoprotein EScience, 1992
- Severe hypercholesterolemia and atherosclerosis in apolipoprotein E-deficient mice created by homologous recombination in ES cellsCell, 1992
- Glycation induces expansion of the molecular packing of collagenJournal of Molecular Biology, 1988