TNF-α is not the cause of fatty liver disease in obese diabetic mice
- 1 January 2001
- journal article
- research article
- Published by Springer Science and Business Media LLC in Nature Medicine
- Vol. 7 (1), 2
- https://doi.org/10.1038/83316
Abstract
No abstract availableThis publication has 12 references indexed in Scilit:
- Metformin reverses fatty liver disease in obese, leptin-deficient miceNature Medicine, 2000
- Mitochondrial Adaptations to Obesity-Related Oxidant StressArchives of Biochemistry and Biophysics, 2000
- Essential role of tumor necrosis factor α in alcohol-induced liver injury in miceGastroenterology, 1999
- Nonalcoholic fatty liver disease: A spectrum of clinical and pathological severity☆, ☆☆Gastroenterology, 1999
- Obesity Induces Expression of Uncoupling Protein-2 in Hepatocytes and Promotes Liver ATP DepletionPublished by Elsevier BV ,1999
- Functional Analysis of Tumor Necrosis Factor (TNF) Receptors in TNF-α-Mediated Insulin Resistance in Genetic Obesity**This work is supported in part by a grant from the NIH (DK-52539).Endocrinology, 1998
- Bacterial Lipopolysaccharide Induces Uncoupling Protein-2 Expression in Hepatocytes by a Tumor Necrosis Factor-α-Dependent MechanismBiochemical and Biophysical Research Communications, 1998
- Kupffer Cells Are a Dominant Site of Uncoupling Protein 2 Expression in Rat LiverBiochemical and Biophysical Research Communications, 1997
- Obesity increases sensitivity to endotoxin liver injury: Implications for the pathogenesis of steatohepatitisProceedings of the National Academy of Sciences of the United States of America, 1997
- Role of uncoupled and non-coupled oxidations in maintenance of safely low levels of oxygen and its one-electron reductantsQuarterly Reviews of Biophysics, 1996