STAT3 Protein Promotes T-cell Survival and Inhibits Interleukin-2 Production through Up-regulation of Class O Forkhead Transcription Factors
Open Access
- 1 September 2011
- journal article
- Published by Elsevier BV in Journal of Biological Chemistry
- Vol. 286 (35), 30888-30897
- https://doi.org/10.1074/jbc.m111.253500
Abstract
No abstract availableKeywords
This publication has 32 references indexed in Scilit:
- Diverse Targets of the Transcription Factor STAT3 Contribute to T Cell Pathogenicity and HomeostasisImmunity, 2010
- Lineage-Specific Biology Revealed by a Finished Genome Assembly of the MousePLoS Biology, 2009
- IL-17 and Th17 CellsAnnual Review of Immunology, 2009
- The Transcription of FOXO Genes Is Stimulated by FOXO3 and Repressed by Growth FactorsJournal of Biological Chemistry, 2009
- The interleukin 23 receptor is essential for the terminal differentiation of interleukin 17–producing effector T helper cells in vivoNature Immunology, 2009
- TH17 cells in development: an updated view of their molecular identity and genetic programmingNature Reviews Immunology, 2008
- Unphosphorylated STAT3 accumulates in response to IL-6 and activates transcription by binding to NFκBGenes & Development, 2007
- TH17 cells contribute to uveitis and scleritis and are expanded by IL-2 and inhibited by IL-27/STAT1Nature Medicine, 2007
- Helper T cell IL-2 production is limited by negative feedback and STAT-dependent cytokine signalsThe Journal of Experimental Medicine, 2007
- DNA Binding Specificity of Different STAT ProteinsJournal of Biological Chemistry, 2001