Mechanisms of Disease: psychomotor retardation and high T3 levels caused by mutations in monocarboxylate transporter 8
- 1 September 2006
- journal article
- review article
- Published by Springer Science and Business Media LLC in Nature Clinical Practice Endocrinology & Metabolism
- Vol. 2 (9), 512-523
- https://doi.org/10.1038/ncpendmet0262
Abstract
No abstract availableKeywords
This publication has 50 references indexed in Scilit:
- Type 2 iodothyronine deiodinase is the major source of plasma T3 in euthyroid humansJCI Insight, 2005
- Role of thyroid hormone during early brain developmentActa Endocrinologica, 2004
- Iodothyronine Levels in the Human Developing Brain: Major Regulatory Roles of Iodothyronine Deiodinases in Different AreasJournal of Clinical Endocrinology & Metabolism, 2004
- Both Thyroid Hormone Receptor (TR)β1 and TRβ2 Isoforms Contribute to the Regulation of Hypothalamic Thyrotropin-Releasing HormoneEndocrinology, 2004
- Mechanisms of thyroid hormone receptor-specific nuclear and extra nuclear actionsMolecular and Cellular Endocrinology, 2003
- Dominant Inhibition of Thyroid Hormone Action Selectively in the Pituitary of Thyroid Hormone Receptor-β Null Mice Abolishes the Regulation of Thyrotropin by Thyroid HormoneMolecular Endocrinology, 2003
- Action of thyroid hormone in brainJournal of Endocrinological Investigation, 2002
- Biochemistry, Cellular and Molecular Biology, and Physiological Roles of the Iodothyronine SelenodeiodinasesEndocrine Reviews, 2002
- Iodothyronine DeiodinasesMethods in enzymology, 2002
- Physiological and Molecular Basis of Thyroid Hormone ActionPhysiological Reviews, 2001