Hypothesis: Antigen-specific T cells prime central nervous system endothelium for recruitment of nonspecific inflammatory cells to effect autoimmune demyelination
- 30 September 1991
- journal article
- review article
- Published by Elsevier BV in Journal of Neuroimmunology
- Vol. 33 (3), 237-244
- https://doi.org/10.1016/0165-5728(91)90111-j
Abstract
No abstract availableThis publication has 36 references indexed in Scilit:
- Adhesion molecules on endothelial cells in the central nervous system: An emerging area in the neuroimmunology of multiple sclerosisClinical Immunology and Immunopathology, 1990
- Shared Human T Cell Receptor V β Usage to Immunodominant Regions of Myelin Basic ProteinScience, 1990
- MALA‐2, mouse homologue of human adhesion molecule ICAM‐1 (CD54)European Journal of Immunology, 1989
- Control of Experimental Autoimmune Encephalomyelitis by T Cells Responding to Activated T CellsScience, 1989
- Perivascular Microglial Cells of the CNS Are Bone Marrow-Derived and Present Antigen in VivoScience, 1988
- Transfer of experimental allergic encephalomyelitis to bone marrow chimeras. Endothelial cells are not a restricting element.The Journal of Experimental Medicine, 1987
- Integrins: A family of cell surface receptorsCell, 1987
- Cellular immune reactivity within the CNSTrends in Neurosciences, 1986
- Coated pits, coated vesicles, and receptor-mediated endocytosisNature, 1979
- THE NATURE AND THE SPECIFICITY OF MONONUCLEAR CELLS IN EXPERIMENTAL AUTOIMMUNE INFLAMMATIONS AND THE MECHANISMS LEADING TO THEIR ACCUMULATIONThe Journal of Experimental Medicine, 1971