Increased expression of PPARγ in high fat diet-induced liver steatosis in mice
- 14 October 2005
- journal article
- Published by Elsevier BV in Biochemical and Biophysical Research Communications
- Vol. 336 (1), 215-222
- https://doi.org/10.1016/j.bbrc.2005.08.070
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
This publication has 31 references indexed in Scilit:
- A novel aminosterol reverses diabetes and fatty liver disease in obese miceJournal of Hepatology, 2004
- CREB controls hepatic lipid metabolism through nuclear hormone receptor PPAR-γNature, 2003
- Growth arrest by troglitazone is mediated by p27Kip1 accumulation, which results from dual inhibition of proteasome activity and Skp2 expression in human hepatocellular carcinoma cellsInternational Journal of Cancer, 2003
- Diabetic KKAy mice exhibit increased hepatic PPARγ1 gene expression and develop hepatic steatosis upon chronic treatment with antidiabetic thiazolidinedionesJournal of Hepatology, 2001
- Adipose tissue is required for the antidiabetic, but not for the hypolipidemic, effect of thiazolidinedionesJCI Insight, 2000
- Activation of PPARγ inhibits cell growth and induces apoptosis in human gastric cancer cellsFEBS Letters, 1999
- Peroxisome Proliferator-activated Receptor γ Ligands Are Potent Inhibitors of Angiogenesis in Vitro and in VivoOnline Journal of Public Health Informatics, 1999
- Activation of PPARγ leads to inhibition of anchorage-independent growth of human colorectal cancer cellsGastroenterology, 1998
- Troglitazone action is independent of adipose tissue.JCI Insight, 1997
- mPPAR gamma 2: tissue-specific regulator of an adipocyte enhancer.Genes & Development, 1994