PTX3, Anti-PTX3, and Anti-C1q Autoantibodies in Lupus Glomerulonephritis
- 25 March 2015
- journal article
- research article
- Published by Springer Science and Business Media LLC in Clinical Reviews in Allergy & Immunology
- Vol. 49 (2), 217-226
- https://doi.org/10.1007/s12016-015-8476-9
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Optimizing outcome in SLE: treating-to-target and definition of treatment goalsAutoimmunity Reviews, 2014
- Involvement of the Tubular ClC-Type Exchanger ClC-5 in Glomeruli of Human Proteinuric NephropathiesPLOS ONE, 2012
- Impaired endothelial function and elevated levels of pentraxin 3 in early‐onset preeclampsiaActa Obstetricia et Gynecologica Scandinavica, 2011
- C1q Deficiency Leads to the Defective Suppression of IFN-α in Response to Nucleoprotein Containing Immune ComplexesThe Journal of Immunology, 2010
- Anti-C1q autoantibodies do not correlate with the occurrence or severity of experimental lupus nephritisNephrology Dialysis Transplantation, 2010
- The role of defective clearance of apoptotic cells in systemic autoimmunityNature Reviews Rheumatology, 2010
- Autoantibodies against Protective Molecules—C1q, C‐Reactive Protein, Serum Amyloid P, Mannose‐Binding Lectin, and Apolipoprotein A1Annals of the New York Academy of Sciences, 2007
- Long-Term Prognosis and Causes of Death in Systemic Lupus ErythematosusAmerican Journal Of Medicine, 2006
- Multimer Formation and Ligand Recognition by the Long Pentraxin PTX3Journal of Biological Chemistry, 1997
- Disease activity in systemic lupus erythematosus: report of the Consensus Study Group of the European Workshop for Rheumatology Research. III. Development of a computerised clinical chart and its application to the comparison of different indices of disease activity. The European Consensus Study Group for Disease Activity in SLE.1992