Midazolam inhibits long-term potentiation through modulation of GABAA receptors
- 31 March 1996
- journal article
- research article
- Published by Elsevier BV in Neuropharmacology
- Vol. 35 (3), 347-357
- https://doi.org/10.1016/0028-3908(95)00182-4
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Loss of synaptic inhibition during repetitive stimulation in genetically epilepsy-prone rats (GEPR)Epilepsy Research, 1994
- Benzodiazepines block long-term potentiation in slices of hippocampus and piriform cortexNeuroscience, 1992
- GABAB autoreceptors regulate the induction of LTPNature, 1991
- Frequency‐dependent depression of inhibition in guinea‐pig neocortex in vitro by GABAB receptor feed‐back on GABA release.The Journal of Physiology, 1989
- Regulation of Glutamate and Aspartate Release from Slices of the Hippocampal CA1 Area: Effects of Adenosine and BaclofenJournal of Neurochemistry, 1988
- Long‐term potentiation involves enhanced synaptic excitation relative to synaptic inhibition in guinea‐pig hippocampus.The Journal of Physiology, 1987
- Dissimilarities between benzodiazepine-binding sites and adenosine uptake sites in astrocytes and neurons in primary culturesJournal of Neuroscience Research, 1987
- Modulation of neurotransmitter action: control of the gamma- aminobutyric acid response through the benzodiazepine receptorJournal of Neuroscience, 1985
- Characteristics of a slow hyperpolarizing synaptic potential in rat hippocampal pyramidal cells in vitroJournal of Neurophysiology, 1984
- Amnesic action of midazolamAnaesthesia, 1980