Mitochondrial involvement in genetically determined transition metal toxicity: II. Copper toxicity
- 27 October 2006
- journal article
- review article
- Published by Elsevier BV in Chemico-Biological Interactions
- Vol. 163 (1-2), 77-85
- https://doi.org/10.1016/j.cbi.2006.05.011
Abstract
No abstract availableKeywords
This publication has 58 references indexed in Scilit:
- Iron and copper metabolismMolecular Aspects of Medicine, 2005
- Cu, Zn-Superoxide Dismutase is Lower and Copper Chaperone CCS is Higher in Erythrocytes of Copper-Deficient Rats and MiceExperimental Biology and Medicine, 2004
- Intracellular Copper Transport in MammalsJournal of Nutrition, 2004
- Mitochondrial dysfunction in neurodegenerative diseases associated with copper imbalance.Neurochemical Research, 2004
- Copper Modulates the Degradation of Copper Chaperone for Cu,Zn Superoxide Dismutase by the 26 S ProteosomeOnline Journal of Public Health Informatics, 2003
- Decrease of cytochrome c oxidase protein in heart mitochondria of copper-deficient ratsBioMetals, 1998
- Newer Findings on a Unified Perspective of Copper Restriction and CardiomyopathyExperimental Biology and Medicine, 1997
- Biologically relevant metal ion‐dependent hydroxyl radical generation An updateFEBS Letters, 1992
- [1] Role of free radicals and catalytic metal ions in human disease: An overviewMethods in Enzymology, 1990
- Studies on lipid peroxidation in rat liver by copper deficiencyInternational Journal of Biochemistry, 1981