The Glucocorticoid Hypothesis of Age‐Related Hippocampal Neurodegeneration: Role of Dysregulated Intraneuronal Calcium
- 1 November 1994
- journal article
- review article
- Published by Wiley in Annals of the New York Academy of Sciences
- Vol. 746 (1), 308-321
- https://doi.org/10.1111/j.1749-6632.1994.tb39249.x
Abstract
No abstract availableKeywords
This publication has 11 references indexed in Scilit:
- Low Ba2+ and Ca2+ induce a sustained high probability of repolarization openings of L-type Ca2+ channels in hippocampal neurons: physiological implications.Proceedings of the National Academy of Sciences of the United States of America, 1993
- Hippocampal glucocorticoid receptor activation enhances voltage-dependent Ca2+ conductances: relevance to brain aging.Proceedings of the National Academy of Sciences of the United States of America, 1992
- Ca2+ channels in rat central and peripheral neurons: High-threshold current resistant to dihydropyridine blockers and ω-conotoxinNeuron, 1991
- Aging-related prolongation of calcium spike duration in rat hippocampal slice neuronsBrain Research, 1990
- Impaired up-regulation of type II corticosteroid receptors in hippocampus of aged ratsBrain Research, 1989
- Calcium and the aging nervous systemNeurobiology of Aging, 1987
- Neuropathology of the Aging Brain and Dementia of the Alzheimer TypePublished by Springer Science and Business Media LLC ,1985
- Prolonged Ca 2+ -Dependent Afterhyperpolarizations in Hippocampal Neurons of Aged RatsScience, 1984
- An Endocrine Hypothesis of Brain Aging and Studies on Brain-Endocrine Correlations and Monosynaptic Neurophysiology during AgingAdvances in experimental medicine and biology, 1978
- The Hippocampus and the Pituitary-Adrenal System HormonesPublished by Springer Science and Business Media LLC ,1975