The induction of CYP1A1 by oltipraz is mediated through calcium-dependent-calpain
- 10 October 2006
- journal article
- Published by Elsevier BV in Toxicology Letters
- Vol. 166 (2), 150-159
- https://doi.org/10.1016/j.toxlet.2006.06.645
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Caspase and calpain function in cell death: bridging the gap between apoptosis and necrosisAnnals of Clinical Biochemistry: International Journal of Laboratory Medicine, 2005
- Calpain Activity Is Generally Elevated during Transformation but Has Oncogene-Specific Biological FunctionsNeoplasia, 2004
- Transcriptional Induction of CYP1A1 by Oltipraz in Human Caco-2 Cells Is Aryl Hydrocarbon Receptor- and Calcium-dependentOnline Journal of Public Health Informatics, 2002
- v-Src-Induced Modulation of the Calpain-Calpastatin Proteolytic System Regulates TransformationMolecular and Cellular Biology, 2002
- Membrane Proximal ERK Signaling Is Required for M-calpain Activation Downstream of Epidermal Growth Factor Receptor SignalingOnline Journal of Public Health Informatics, 2001
- The hsp90 Chaperone Complex Regulates Intracellular Localization of the Dioxin ReceptorMolecular and Cellular Biology, 2001
- Aryl Hydrocarbon Receptor Imported into the Nucleus following Ligand Binding Is Rapidly Degraded via the Cytosplasmic Proteasome following Nuclear ExportJournal of Biological Chemistry, 1999
- Prolonged depletion of AH receptor without alteration of receptor mRNA levels after treatment of cells in culture with 2,3,7,8-tetrachlorodibenzo-p-dioxinBiochemical Pharmacology, 1998
- Nuclear Localization and Export Signals of the Human Aryl Hydrocarbon ReceptorOnline Journal of Public Health Informatics, 1998
- Control of Cytochrome P 1 -450 Gene Expression by DioxinScience, 1985