Protofibrils of amyloid β-protein inhibit specific K+ currents in neocortical cultures
- 27 June 2003
- journal article
- Published by Elsevier BV in Neurobiology of Disease
- Vol. 13 (3), 177-190
- https://doi.org/10.1016/s0969-9961(03)00068-8
Abstract
No abstract availableThis publication has 35 references indexed in Scilit:
- Immunization reverses memory deficits without reducing brain Aβ burden in Alzheimer's disease modelNature Neuroscience, 2002
- Naturally secreted oligomers of amyloid β protein potently inhibit hippocampal long-term potentiation in vivoNature, 2002
- The 'Arctic' APP mutation (E693G) causes Alzheimer's disease by enhanced Aβ protofibril formationNature Neuroscience, 2001
- Soluble Amyloid β Peptide Concentration as a Predictor of Synaptic Change in Alzheimer's DiseaseThe American Journal of Pathology, 1999
- Amyloid β-Protein FibrillogenesisJournal of Biological Chemistry, 1999
- Amyloid β-Protein FibrillogenesisJournal of Biological Chemistry, 1997
- Soluble β-Amyloid Induction of Alzheimer's Phenotype for Human Fibroblast K + ChannelsScience, 1994
- Hippocampal damage produced in rats by α-dendrotoxin—a selective K+ channel blocker—involves non-NMDA receptor activationNeurochemistry International, 1994
- Excitotoxicity induced by enhanced excitatory neurotransmission in cultured hippocampal pyramidal neuronsNeuron, 1990
- A family of calcium-dependent potassium channels from rat brainNeuron, 1989