High Levels of Circulating Aβ42 Are Sequestered by Plasma Proteins in Alzheimer's Disease
- 21 April 1999
- journal article
- Published by Elsevier BV in Biochemical and Biophysical Research Communications
- Vol. 257 (3), 787-791
- https://doi.org/10.1006/bbrc.1999.0552
Abstract
No abstract availableKeywords
This publication has 28 references indexed in Scilit:
- Elevated Low-Density Lipoprotein in Alzheimer's Disease Correlates with Brain Aβ 1–42 LevelsBiochemical and Biophysical Research Communications, 1998
- Increased plasma amyloid β protein 1–42 levels in Down syndromeNeuroscience Letters, 1998
- Cerebrovascular Accumulation and Increased Blood‐Brain Barrier Permeability to Circulating Alzheimer's Amyloid β Peptide in Aged Squirrel Monkey with Cerebral Amyloid AngiopathyJournal of Neurochemistry, 1998
- Isolation, Chemical Characterization, and Quantitation of Aβ 3-Pyroglutamyl Peptide from Neuritic Plaques and Vascular Amyloid DepositsBiochemical and Biophysical Research Communications, 1997
- α2‐Macroglobulin as a β‐Amyloid Peptide‐Binding Plasma ProteinJournal of Neurochemistry, 1997
- Complement interactions with amyloid β1–42: A nidus for inflammation in AD brainsAmyloid, 1997
- Biochemical Characterization of Alzheimer's Soluble Amyloid Beta Protein in Human Cerebrospinal Fluid: Association with High Density LipoproteinsBiochemical and Biophysical Research Communications, 1996
- 15-year longitudinal study of blood pressure and dementiaThe Lancet, 1996
- Amyloid β‐Protein Induces Its Own Production in Cultured Degenerating Cerebrovascular Smooth Muscle CellsJournal of Neurochemistry, 1995
- Characterization of Apolipoprotein J-Alzheimer's Aβ InteractionOnline Journal of Public Health Informatics, 1995