Inhibition of Proinflammatory RANTES Expression by TGF-β1 Is Mediated by Glycogen Synthase Kinase-3β-dependent β-Catenin Signaling
Open Access
- 1 March 2011
- journal article
- Published by Elsevier BV
- Vol. 286 (9), 7052-7059
- https://doi.org/10.1074/jbc.m110.174821
Abstract
No abstract availableThis publication has 40 references indexed in Scilit:
- Plasminogen Activator Inhibitor-1 Is a Transcriptional Target of the Canonical Pathway of Wnt/β-Catenin SignalingPublished by Elsevier BV ,2010
- New Insights into Epithelial-Mesenchymal Transition in Kidney FibrosisJournal of the American Society of Nephrology, 2010
- Wnt/β-Catenin Signaling Promotes Podocyte Dysfunction and AlbuminuriaJournal of the American Society of Nephrology, 2009
- Increased IκBα expression is essential for the tolerogenic property of TGF‐β‐exposed APCSThe FASEB Journal, 2009
- Paricalcitol Inhibits Renal Inflammation by Promoting Vitamin D Receptor–Mediated Sequestration of NF-κB SignalingJournal of the American Society of Nephrology, 2008
- Hepatocyte Growth Factor Exerts Its Anti-Inflammatory Action by Disrupting Nuclear Factor-κB SignalingThe American Journal of Pathology, 2008
- Hepatocyte Growth Factor Suppresses Proinflammatory NFκB Activation through GSK3β Inactivation in Renal Tubular Epithelial CellsPublished by Elsevier BV ,2008
- Dose-dependent cross-talk between the transforming growth factor-β and interleukin-1 signaling pathwaysProceedings of the National Academy of Sciences of the United States of America, 2007
- Mechanisms of Disease: regulation of RANTES (CCL5) in renal diseaseNature Clinical Practice Nephrology, 2007
- Chemokines and chemokine receptors are involved in the resolution or progression of renal diseaseKidney International, 2003