Downstream Activation of a TATA-less Promoter by Oct-2, Bob1, and NF-κB Directs Expression of the Homing Receptor BLR1 to Mature B Cells
Open Access
- 1 November 1998
- journal article
- Published by Elsevier BV
- Vol. 273 (44), 28831-28836
- https://doi.org/10.1074/jbc.273.44.28831
Abstract
No abstract availableThis publication has 31 references indexed in Scilit:
- Nuclear Factor (NF)-κB2 (p100/p52) Is Required for Normal Splenic Microarchitecture and B Cell–mediated Immune ResponsesThe Journal of Experimental Medicine, 1998
- Requirement for NF-κB in osteoclast and B-cell developmentGenes & Development, 1997
- B lymphocytes are impaired in mice lacking the transcriptional co‐activator Bob1/OCA‐B/OBF1European Journal of Immunology, 1996
- The B-cell-specific transcription coactivator OCA-B/OBF-1/Bob-1 is essential for normal production of immunoglobulin isotypesNature, 1996
- B-cell-specif ic coactivator OBF-1/OCA-B/Bob1 required for immune response and germinal centre formationNature, 1996
- Functional Characterization of the Murine Homolog of the B cell-specific Coactivator BOB.1/OBF.1Published by Elsevier BV ,1995
- The G protein‐coupled receptor BLR1 is involved in murine B cell differentiation and is also expressed in neuronal tissuesEuropean Journal of Immunology, 1993
- Cloning of a novel putative G‐protein‐coupled receptor (NLR) which is expressed in neuronal and lymphatic tissueFEBS Letters, 1993
- Differentiation‐specific expression of a novel G protein‐coupled receptor from Burkitt's lymphomaEuropean Journal of Immunology, 1992
- The “initiator” as a transcription control elementCell, 1989