Six1 and Six4 are essential for Gdnf expression in the metanephric mesenchyme and ureteric bud formation, while Six1 deficiency alone causes mesonephric-tubule defects
- 30 April 2007
- journal article
- Published by Elsevier BV in Mechanisms of Development
- Vol. 124 (4), 290-303
- https://doi.org/10.1016/j.mod.2007.01.002
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Health, Labour and Welfare
- Japan Society for the Promotion of Science
- Ministry of Education, Culture, Sports, Science and Technology
This publication has 51 references indexed in Scilit:
- Wnt9b Plays a Central Role in the Regulation of Mesenchymal to Epithelial Transitions Underlying Organogenesis of the Mammalian Urogenital SystemDevelopmental Cell, 2005
- Slc12a2 is a direct target of two closely related homeobox proteins, Six1 and Six4The FEBS Journal, 2005
- Six1 is not involved in limb tendon development, but is expressed in limb connective tissue under Shh regulationMechanisms of Development, 2005
- The transcription factor Six2 activates expression of the Gdnf gene as well as its own promoterMechanisms of Development, 2004
- Nephric lineage specification by Pax2 and Pax8Genes & Development, 2002
- Regulation of ureteric bud outgrowth by Pax2-dependent activation of the glial derived neurotrophic factor geneDevelopment, 2001
- GFRα1-Deficient Mice Have Deficits in the Enteric Nervous System and KidneysNeuron, 1998
- GFRα1 Is an Essential Receptor Component for GDNF in the Developing Nervous System and KidneyNeuron, 1998
- GDNF signalling through the Ret receptor tyrosine kinaseNature, 1996
- Deletion 14q (q22q23) associated with anophthalmia, absent pituitary, and other abnormalities.Journal of Medical Genetics, 1991