Regulation by interferon β-1a of reactive oxygen metabolites production by lymphocytes and monocytes and serum sulfhydryls in relapsing multiple sclerosis patients
- 31 January 2003
- journal article
- Published by Elsevier BV in Neurochemistry International
- Vol. 42 (1), 67-71
- https://doi.org/10.1016/s0197-0186(02)00057-8
Abstract
No abstract availableKeywords
This publication has 13 references indexed in Scilit:
- Effect of early interferon treatment on conversion to definite multiple sclerosis: a randomised studyThe Lancet, 2001
- Intramuscular Interferon Beta-1A Therapy Initiated during a First Demyelinating Event in Multiple SclerosisNew England Journal of Medicine, 2000
- The oxidation-reduction state of serum proteins in multiple sclerosis patients: effect of Interferon beta-1bNeurochemistry International, 1999
- Oxidative damage to DNA in plaques of MS brainsMultiple Sclerosis Journal, 1998
- Rapid Communication Interferon beta-1b inhibits reactive oxygen species production in peripheral blood monocytes of patients with relapsing-remitting multiple sclerosisNeurochemistry International, 1998
- The role of reactive oxygen species in the pathogenesis of multiple sclerosisMedical Hypotheses, 1992
- Regulation of microglial function by interferonsJournal of Neuroimmunology, 1992
- Oxygen radicals and the nervous systemTrends in Neurosciences, 1985
- Effect of Reactive Oxygen Species on Myelin Membrane ProteinsJournal of Neurochemistry, 1985
- Serum Sulfhydryl Determinations and Significance in Connective Tissue DiseasesAnnals of Internal Medicine, 1964