Deletion of the Mint3/Apba3 Gene in Mice Abrogates Macrophage Functions and Increases Resistance to Lipopolysaccharide-induced Septic Shock
Open Access
- 1 September 2011
- journal article
- Published by Elsevier BV in Online Journal of Public Health Informatics
- Vol. 286 (37), 32542-32551
- https://doi.org/10.1074/jbc.m111.271726
Abstract
No abstract availableThis publication has 33 references indexed in Scilit:
- Hypoxia-inducible factor 2α regulates macrophage function in mouse models of acute and tumor inflammationJCI Insight, 2010
- Matrix Metalloproteinases: Regulators of the Tumor MicroenvironmentCell, 2010
- HIF-1: upstream and downstream of cancer metabolismCurrent Opinion in Genetics & Development, 2010
- Induction of a MT1-MMP and MT2-MMP-dependent basement membrane transmigration program in cancer cells by Snail1Proceedings of the National Academy of Sciences of the United States of America, 2009
- Mint3 Enhances the Activity of Hypoxia-inducible Factor-1 (HIF-1) in Macrophages by Suppressing the Activity of Factor Inhibiting HIF-1Online Journal of Public Health Informatics, 2009
- Cytoplasmic tail of MT1‐MMP regulates macrophage motility independently from its protease activityGenes to Cells, 2009
- Trophic macrophages in development and diseaseNature Reviews Immunology, 2009
- Hypoxia, HIF1 and glucose metabolism in the solid tumourNature Reviews Cancer, 2008
- Interaction of Mint3 with Furin regulates the localization of Furin in the trans-Golgi networkJournal of Cell Science, 2008
- Oxygen Sensing by Metazoans: The Central Role of the HIF Hydroxylase PathwayMolecular Cell, 2008