Mig6 Is a Sensor of EGF Receptor Inactivation that Directly Activates c-Abl to Induce Apoptosis during Epithelial Homeostasis
Open Access
- 11 September 2012
- journal article
- Published by Elsevier BV in Developmental Cell
- Vol. 23 (3), 547-559
- https://doi.org/10.1016/j.devcel.2012.08.001
Abstract
No abstract availableKeywords
This publication has 36 references indexed in Scilit:
- Tumor Necrosis Factor Alpha-Induced Apoptosis Requires p73 and c-ABL Activation Downstream of RB DegradationMolecular and Cellular Biology, 2004
- Apoptosis and Dependence Receptors: A Molecular Basis for Cellular AddictionPhysiological Reviews, 2004
- Tissue-Wide Expression Profiling Using cDNA Subtraction and Microarrays to Identify Tumor-Specific GenesCancer Research, 2004
- The dependence receptor hypothesisApoptosis, 2004
- Regulation of the c-Abl and Bcr–Abl tyrosine kinasesNature Reviews Molecular Cell Biology, 2004
- Profiling of tyrosine phosphorylation pathways in human cells using mass spectrometryProceedings of the National Academy of Sciences of the United States of America, 2003
- The Role of Apoptosis in Creating and Maintaining Luminal Space within Normal and Oncogene-Expressing Mammary AciniCell, 2002
- Targeting of the c-Abl Tyrosine Kinase to Mitochondria in Endoplasmic Reticulum Stress-Induced ApoptosisMolecular and Cellular Biology, 2001
- ErbB2, but not ErbB1, reinitiates proliferation and induces luminal repopulation in epithelial aciniNature, 2001
- Neonatal lethality and lymphopenia in mice with a homozygous disruption of the c-abl proto-oncogeneCell, 1991