Effects of individual segmental trisomies of human chromosome 21 syntenic regions on hippocampal long-term potentiation and cognitive behaviors in mice
- 17 December 2010
- journal article
- Published by Elsevier BV in Brain Research
- Vol. 1366, 162-171
- https://doi.org/10.1016/j.brainres.2010.09.107
Abstract
No abstract availableKeywords
Funding Information
- Roswell Park Alliance Foundation
- Louis Sklarow Memorial Fund
- Fondation Jérôme Lejeune
- Children's Guild Foundation
- NIH (R01HL091519, R01NS066072, P30CA016056)
This publication has 59 references indexed in Scilit:
- Olig1 and Olig2 triplication causes developmental brain defects in Down syndromeNature Neuroscience, 2010
- A mouse model of Down syndrome trisomic for all human chromosome 21 syntenic regionsHuman Molecular Genetics, 2010
- The role of SAP97 in synaptic glutamate receptor dynamicsProceedings of the National Academy of Sciences of the United States of America, 2010
- Behavioral deficits and subregion-specific suppression of LTP in mice expressing a population of mutant NMDA receptors throughout the hippocampusLearning & Memory, 2009
- A new mouse model for the trisomy of the Abcg1–U2af1 region reveals the complexity of the combinatorial genetic code of down syndromeHuman Molecular Genetics, 2009
- G protein-activated inwardly rectifying potassium channels mediate depotentiation of long-term potentiationProceedings of the National Academy of Sciences of the United States of America, 2009
- NFAT dysregulation by increased dosage of DSCR1 and DYRK1A on chromosome 21Nature, 2006
- Effects of the rd1 Mutation and Host Strain on Hippocampal Learning in MiceBehavior Genetics, 2005
- The neuropsychology of mental retardationJournal of the International Neuropsychological Society, 1996
- A behavioral assessment of Ts65Dn mice: a putative Down syndrome modelNeuroscience Letters, 1995