Changes in Protein Kinase C and its Presynaptic Substrate B-SO/GAP-43 after Intrauterine Exposure to Methylazoxy-methanol, a Treatment Inducing Cortical and Hippocampal Damage and Cognitive Deficit in Rats
- 1 May 1995
- journal article
- Published by Wiley in European Journal of Neuroscience
- Vol. 7 (5), 899-906
- https://doi.org/10.1111/j.1460-9568.1995.tb01077.x
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Selective in vitro blockade of neuroepithelial cells proliferation by methylazoxymethanol, a molecule capable of inducing long lasting functional impairmentsJournal of Neuroscience Research, 1995
- Purification of B-50 by 2-mercaptoethanol extraction from rat brain synaptosomal plasma membranesNeurochemical Research, 1993
- Protein kinase C modulation of NMDA currents: an important link for LTP inductionTrends in Neurosciences, 1992
- Microencephaly reduces the phosphorylation of the PKC substrate B-50/GAP43 in rat cortex and hippocampusBrain Research, 1991
- Chapter 13: Protein kinase C substrate B-50 (GAP-43) and neurotransmitter releaseProgress in Brain Research, 1991
- Evidence for the Binding of Calmodulin to Endogenous B‐50 (GAP‐43) in Native Synaptosomal Plasma MembranesJournal of Neurochemistry, 1990
- Inhibition of noradrenaline release by antibodies to B-50 (GAP-43)Nature, 1989
- Determination of Changes in the Phosphorylation State of the Neuron‐Specific Protein Kinase C Substrate B‐50 (GAP43) by Quantitative ImmunoprecipitationJournal of Neurochemistry, 1989
- Distinct patterns of expression of different protein kinase C mRNAs in rat tissuesCell, 1987
- Fetally-induced noradrenergic hyperinnervation of cerebral cortex results in persistent down-regulation of beta-receptorsDevelopmental Brain Research, 1982