Environmental Copper and Manganese in the Pathophysiology of Neurologic Diseases (Alzheimer's Disease and Manganism)
- 11 April 2005
- journal article
- review article
- Published by Wiley in Acta Hydrochimica et Hydrobiologica
- Vol. 33 (1), 72-78
- https://doi.org/10.1002/aheh.200400556
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- In vivo reduction of amyloid-β by a mutant copper transporterProceedings of the National Academy of Sciences of the United States of America, 2003
- The metallobiology of Alzheimer's diseaseTrends in Neurosciences, 2003
- Overexpression of Alzheimer's Disease Amyloid-β Opposes the Age-dependent Elevations of Brain Copper and IronJournal of Biological Chemistry, 2002
- Possible mechanisms of APP-mediated oxidative stress in Alzheimer’s disease1,2 1Guest Editors: Mark A. Smith and George Perry 2This article is part of a series of reviews on “Causes and Consequences of Oxidative Stress in Alzheimer’s Disease.” The full list of papers may be found on the homepage of the journal.Free Radical Biology & Medicine, 2002
- Characterization of Copper Interactions with Alzheimer Amyloid β PeptidesJournal of Neurochemistry, 2000
- Copper inhibits β-amyloid production and stimulates the non-amyloidogenic pathway of amyloid-precursor-protein secretionBiochemical Journal, 1999
- It all sticks together—the APP-related family of proteins and Alzheimer's diseaseMolecular Psychiatry, 1999
- Copper levels are increased in the cerebral cortex and liver of APP and APLP2 knockout miceBrain Research, 1999
- The Amyloid Precursor Protein of Alzheimer's Disease in the Reduction of Copper(II) to Copper(I)Science, 1996
- The βA4 amyloid precursor protein binding to copperFEBS Letters, 1994