Pancreatic β-Cell-specific Repression of Insulin Gene Transcription by CCAAT/Enhancer-binding Protein β
Open Access
- 1 November 1997
- journal article
- Published by Elsevier BV
- Vol. 272 (45), 28349-28359
- https://doi.org/10.1074/jbc.272.45.28349
Abstract
No abstract availableKeywords
This publication has 51 references indexed in Scilit:
- Differentiation of glucose toxicity from beta cell exhaustion during the evolution of defective insulin gene expression in the pancreatic islet cell line, HIT-T15.JCI Insight, 1997
- C/EBP-β/LAP Controls Down-regulation of Albumin Gene Transcription during Liver RegenerationPublished by Elsevier BV ,1996
- Chronic exposure of betaTC-6 cells to supraphysiologic concentrations of glucose decreases binding of the RIPE3b1 insulin gene transcription activator.JCI Insight, 1996
- DNA Binding Specificity of the CCAAT/Enhancer-binding Protein Transcription Factor FamilyPublished by Elsevier BV ,1996
- Pancreatic Islet Expression of the Homeobox Factor STF-1 Relies on an E-box Motif That Binds USFJournal of Biological Chemistry, 1996
- Tissue-specific regulation of the insulin gene by a novel basic helix-loop-helix transcription factor.Genes & Development, 1995
- Protein kinase A and C site-specific phosphorylations of LAP (NF-IL6) modulate its binding affinity to DNA recognition elements.JCI Insight, 1994
- Prolonged exposure of human pancreatic islets to high glucose concentrations in vitro impairs the beta-cell function.JCI Insight, 1992
- Calcium-Regulated Phosphorylation Within the Leucine Zipper of C/EBPβScience, 1992
- Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4Nature, 1970