Understanding the mechanisms for metabolism-linked hemolytic toxicity of primaquine against glucose 6-phosphate dehydrogenase deficient human erythrocytes: Evaluation of eryptotic pathway
- 29 March 2012
- journal article
- Published by Elsevier BV in Toxicology
- Vol. 294 (1), 54-60
- https://doi.org/10.1016/j.tox.2012.01.015
Abstract
No abstract availableKeywords
This publication has 30 references indexed in Scilit:
- Primaquine in vivax malaria: an update and review on management issuesMalaria Journal, 2011
- Epidemiology and Infectivity of Plasmodium falciparum and Plasmodium vivax Gametocytes in Relation to Malaria Control and EliminationClinical Microbiology Reviews, 2011
- The reality of using primaquineMalaria Journal, 2010
- Medications and Glucose-6-Phosphate Dehydrogenase DeficiencyDrug Safety, 2010
- Blood cell oxidative stress precedes hemolysis in whole blood–liver slice co-cultures of rat, dog, and human tissuesToxicology and Applied Pharmacology, 2010
- Targeting the hypnozoite reservoir of Plasmodium vivax: the hidden obstacle to malaria eliminationTrends in Parasitology, 2010
- Membrane Characteristics and Metabolic Properties of Glucose-6-Phosphate Dehydrogenase Deficient Red CellsBritish Journal of Haematology, 2008
- Primaquine-Induced Hemolytic Anemia: Role of Splenic Macrophages in the Fate of 5-Hydroxyprimaquine-Treated Rat ErythrocytesThe Journal of pharmacology and experimental therapeutics, 2005
- Primaquine-Induced Hemolytic Anemia: Role of Membrane Lipid Peroxidation and Cytoskeletal Protein Alterations in the Hemotoxicity of 5-HydroxyprimaquineThe Journal of pharmacology and experimental therapeutics, 2005
- Oxidant Stress and Haemolysis of the Human ErythrocyteToxicological Reviews, 2004