Inhibition of Glycolate Oxidase With Dicer-substrate siRNA Reduces Calcium Oxalate Deposition in a Mouse Model of Primary Hyperoxaluria Type 1
Open Access
- 1 April 2016
- journal article
- Published by Elsevier BV in Molecular Therapy
- Vol. 24 (4), 770-778
- https://doi.org/10.1038/mt.2016.4
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- Hydroxyproline metabolism in mouse models of primary hyperoxaluriaAmerican Journal of Physiology-Renal Physiology, 2012
- Hyperoxalurie primitiveNephrologie & Therapeutique, 2011
- Enteric oxalate elimination is induced and oxalate is normalized in a mouse model of primary hyperoxaluria following intestinal colonization withOxalobacterAmerican Journal of Physiology-Gastrointestinal and Liver Physiology, 2011
- Gene Therapy Moves Forward in 2010Molecular Therapy, 2011
- Primary HyperoxaluriaInternational Journal of Nephrology, 2011
- The Roles and Mechanisms of Intestinal Oxalate Transport in Oxalate HomeostasisSeminars in Nephrology, 2008
- Hydroxyproline ingestion and urinary oxalate and glycolate excretionKidney International, 2006
- Alanine–glyoxylate aminotransferase-deficient mice, a model for primary hyperoxaluria that responds to adenoviral gene transferProceedings of the National Academy of Sciences of the United States of America, 2006
- Treatment of the primary hyperoxalurias: A new chapterKidney International, 2006
- Peroxisomal alanine:glyoxylate aminotransferase deficiency in primary hyperoxaluria type IFEBS Letters, 1986