The Role of 3′ Poly(A) Tail Metabolism in Tumor Necrosis Factor-α Regulation
Open Access
- 1 August 1997
- journal article
- Published by Elsevier BV in Journal of Biological Chemistry
- Vol. 272 (34), 21120-21127
- https://doi.org/10.1074/jbc.272.34.21120
Abstract
No abstract availableKeywords
This publication has 33 references indexed in Scilit:
- The importance of a lipopolysaccharide-initiated, cytokine-mediated host defense mechanism in mice against extraintestinally invasive Escherichia coli.JCI Insight, 1995
- Tumor necrosis factor activities and cancer therapy — A perspectivePharmacology & Therapeutics, 1993
- Cytoplasmic regulation of mRNA function: The importance of the 3′ untranslated regionCell, 1993
- 3'-UTR-dependent deadenylation by the yeast poly(A) nuclease.Genes & Development, 1992
- Endotoxin-responsive sequences control cachectin/tumor necrosis factor biosynthesis at the translational level.The Journal of Experimental Medicine, 1990
- Pretreatment with recombinant murine tumor necrosis factor alpha/cachectin and murine interleukin 1 alpha protects mice from lethal bacterial infection.The Journal of Experimental Medicine, 1989
- Molecular control of tissue‐specific expression at the mouse TNF locusEuropean Journal of Immunology, 1989
- Control of Cachectin (Tumor Necrosis Factor) Synthesis: Mechanisms of Endotoxin ResistanceScience, 1986
- Passive Immunization Against Cachectin/Tumor Necrosis Factor Protects Mice from Lethal Effect of EndotoxinScience, 1985
- Clinical PhysicsScandinavian Journal of Clinical and Laboratory Investigation, 1968