The proline TP53 variant stimulates likely lymphangiogenesis in an orthotopic mouse model of pancreatic cancer
Open Access
- 6 December 2011
- journal article
- Published by Springer Science and Business Media LLC in British Journal of Cancer
- Vol. 106 (2), 348-357
- https://doi.org/10.1038/bjc.2011.521
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- Molecular mechanism of pancreatic cancer—understanding proliferation, invasion, and metastasisLangenbecks Archives Of Surgery, 2010
- Advanced pancreatic carcinoma: current treatment and future challengesNature Reviews Clinical Oncology, 2010
- Polymorphism in wild-type p53 modulates response to chemotherapy in vitro and in vivoOncogene, 2004
- Multiple regulatory pathways of vascular permeability factor/vascular endothelial growth factor (VPF/VEGF) expression in tumorsSeminars in Cancer Biology, 2004
- The hypoxic response of tumors is dependent on their microenvironmentCancer Cell, 2003
- p53 protein stability in tumour cells is not determined by mutation but is dependent on Mdm2 bindingOncogene, 1997
- Tumor-suppressive pathways in pancreatic carcinoma.1997
- Pancreatic carcinomaThe Lancet, 1997
- The p53 Tumor Suppressor Gene Inhibits Angiogenesis by Stimulating the Production of ThrombospondinCold Spring Harbor Symposia on Quantitative Biology, 1994
- Tumor Angiogenesis and Metastasis — Correlation in Invasive Breast CarcinomaNew England Journal of Medicine, 1991