PGC-1α over-expression promotes recovery from mitochondrial dysfunction and cell injury
- 13 April 2007
- journal article
- Published by Elsevier BV in Biochemical and Biophysical Research Communications
- Vol. 355 (3), 734-739
- https://doi.org/10.1016/j.bbrc.2007.02.023
Abstract
No abstract availableKeywords
This publication has 27 references indexed in Scilit:
- Suppression of Reactive Oxygen Species and Neurodegeneration by the PGC-1 Transcriptional CoactivatorsCell, 2006
- Nitric oxide regulates mitochondrial oxidative stress protection via the transcriptional coactivator PGC‐1αThe FASEB Journal, 2006
- Flutamide restores cardiac function after trauma-hemorrhage via an estrogen-dependent pathway through upregulation of PGC-1American Journal of Physiology-Heart and Circulatory Physiology, 2006
- PGC-1 upregulation via estrogen receptors: a common mechanism of salutary effects of estrogen and flutamide on heart function after trauma-hemorrhageAmerican Journal of Physiology-Heart and Circulatory Physiology, 2005
- Selective Mitochondrial Autophagy, or Mitophagy, as a Targeted Defense Against Oxidative Stress, Mitochondrial Dysfunction, and AgingRejuvenation Research, 2005
- Activation of ERK or inhibition of JNK ameliorates H2O2 cytotoxicity in mouse renal proximal tubule cellsKidney International, 2004
- Characterization of Superoxide-producing Sites in Isolated Brain MitochondriaJournal of Biological Chemistry, 2004
- Cisplatin nephrotoxicitySeminars in Nephrology, 2003
- Mechanisms Controlling Mitochondrial Biogenesis and Respiration through the Thermogenic Coactivator PGC-1Cell, 1999
- Metabolism of Isolated Kidney TubulesEuropean Journal of Biochemistry, 1979