SMAD4 is essential for generating subtypes of neurons during cerebellar development
- 1 May 2012
- journal article
- Published by Elsevier BV in Developmental Biology
- Vol. 365 (1), 82-90
- https://doi.org/10.1016/j.ydbio.2012.02.017
Abstract
No abstract availableKeywords
Funding Information
- NIH (MH083804, MH070596)
- McDonnell Brain Tumor Foundation
This publication has 46 references indexed in Scilit:
- Lmx1a regulates fates and location of cells originating from the cerebellar rhombic lip and telencephalic cortical hemProceedings of the National Academy of Sciences of the United States of America, 2010
- TAK1 is an essential regulator of BMP signalling in cartilageThe EMBO Journal, 2009
- IKKα, a critical regulator of epidermal differentiation and a suppressor of skin cancerThe EMBO Journal, 2008
- Ectodermal Smad4 and p38 MAPK Are Functionally Redundant in Mediating TGF-β/BMP Signaling during Tooth and Palate DevelopmentDevelopmental Cell, 2008
- Medulloblastoma Can Be Initiated by Deletion of Patched in Lineage-Restricted Progenitors or Stem CellsCancer Cell, 2008
- Ascl1 (Mash1) lineage cells contribute to discrete cell populations in CNS architectureMolecular and Cellular Neuroscience, 2008
- Specific regions within the embryonic midbrain and cerebellum require different levels of FGF signaling during developmentDevelopment, 2008
- Cerebellar GABAergic progenitors adopt an external granule cell-like phenotype in the absence of Ptf1a transcription factor expressionProceedings of the National Academy of Sciences of the United States of America, 2007
- Bone Morphogenetic ProteinsGrowth Factors, 2004
- Smad-dependent and Smad-independent pathways in TGF-β family signallingNature, 2003