Rapid repair of UVA‐induced oxidized purines and persistence of UVB‐induced dipyrimidine lesions determine the mutagenicity of sunlight in mouse cells
Open Access
- 11 February 2008
- journal article
- research article
- Published by Wiley in The FASEB Journal
- Vol. 22 (7), 2379-2392
- https://doi.org/10.1096/fj.07-105437
Abstract
Despite the predominance of ultraviolet A (UVA) relative to UVB in terrestrial sunlight, solar mutagenesis in humans and rodents is characterized by mutations specific for UVB. We have investigated t...Keywords
Funding Information
- National Institute of Environmental Research (ES06070)
This publication has 127 references indexed in Scilit:
- Riboflavin activated by ultraviolet A1 irradiation induces oxidative DNA damage-mediated mutations inhibited by vitamin CProceedings of the National Academy of Sciences, 2007
- Mutagenicity of ultraviolet A radiation in the lacI transgene in Big Blue mouse embryonic fibroblastsMutation Research, 2007
- Patterns of somatic mutation in human cancer genomesNature, 2007
- The association of use of sunbeds with cutaneous malignant melanoma and other skin cancers: A systematic reviewInternational Journal of Cancer, 2006
- Cyclobutane pyrimidine dimers are predominant DNA lesions in whole human skin exposed to UVA radiationProceedings of the National Academy of Sciences, 2006
- New functions of XPC in the protection of human skin cells from oxidative damageThe EMBO Journal, 2006
- Mutations of the BRAF gene in human cancerNature, 2002
- Preferential repair of ultraviolet light‐induced dna damage in the transcribed strand of the human p53 geneMolecular Carcinogenesis, 1994
- OZONE DEPLETION AND INCREASE IN ANNUAL CARCINOGENIC ULTRAVIOLET DOSEPhotochemistry and Photobiology, 1990
- UV INDUCED (6‐4) PHOTOPRODUCTS ARE DISTRIBUTED DIFFERENTLY THAN CYCLOBUTANE DIMERS IN NUCLEOSOMESPhotochemistry and Photobiology, 1990