The Orphan Nuclear Receptor RORγt Directs the Differentiation Program of Proinflammatory IL-17+ T Helper Cells
Top Cited Papers
- 22 September 2006
- journal article
- Published by Elsevier BV in Cell
- Vol. 126 (6), 1121-1133
- https://doi.org/10.1016/j.cell.2006.07.035
Abstract
No abstract availableKeywords
This publication has 57 references indexed in Scilit:
- The IL-23/IL-17 axis in inflammationJCI Insight, 2006
- Anti-IL-23 therapy inhibits multiple inflammatory pathways and ameliorates autoimmune encephalomyelitisJCI Insight, 2006
- IL-23 is essential for T cell–mediated colitis and promotes inflammation via IL-17 and IL-6JCI Insight, 2006
- Transforming growth factor-β induces development of the TH17 lineageNature, 2006
- Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cellsNature, 2006
- Divergent roles of IL-23 and IL-12 in host defense against Klebsiella pneumoniae The Journal of Experimental Medicine, 2005
- Anatomical basis of tolerance and immunity to intestinal antigensNature Reviews Immunology, 2003
- Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brainNature, 2003
- THE INTERLEUKIN-12/INTERLEUKIN-12-RECEPTOR SYSTEM: Role in Normal and Pathologic Immune ResponsesAnnual Review of Immunology, 1998
- The role of IL-12 in the induction of organ-specific autoimmune diseasesImmunology Today, 1995